Chief Pathology characteristic:Epithelioid cells nodules口Langerhans cellular infiltration口
Chief Pathology characteristic: □ Epithelioid cells nodules □ Langerhans cellular infiltration
Pathological prognosis1）AbsorptionComplete absorption Calcification orScleromaCalcified lesions occur at least 6 to 12 months2) progressionExpand lesionsBronchial lymphfistulaAtelectasisEm physem aTuberculous pleurisy3）DeteriorationHematogenous dissemination
□ Pathological prognosis 1）Absorption Complete absorption、 Calcification orScleroma Calcified lesions occur at least 6 to 12 months 2）progression Expand lesions Bronchial lymphfistula A telectasis、E m physem a Tuberculous pleurisy 3）Deterioration Hematogenous dissemination
Pathologic prognosisPrimarycomplexProgressionHematogenic spreadHealingwithExpansion oflesionsobsorptionoccultDeterioration:cavitas cavitationcalcificationhematogenicacute hemat- tuberculous pleurisyscleromaspreadogenic spreadatelectasis++pulmonaryemphysemaMultiple organsDisseminated口Extension tobronchus-TB: miliary TB,remote focibronchial lymph node fistulaTBM:endobronchial tuberculosiscaseouspneumonia
Hematogenic spread Healing with □ obsorption □ calcification □ scleroma Progression Expansion of lesions Deterioration: acute hematogenic spread occult hematogenic spread Disseminated TB:miliary TB, TBM Multiple organs remote foci □ cavitas cavitation □ tuberculous pleurisy □ atelectasis □ pulmonary emphysema □ Extension to bronchus→ bronchial lymph node fistula • endobronchial tuberculosis • caseous pneumonia Primary complex Pathologic prognosis
灸70calcificationabsorptionsclerom a
absorption sclerom a calcification
cavitastuberculous pleurisyobstructivepulmonaryemphysemacaseouspneumoniaatelectasismiliaryTB
cavitas tuberculous pleurisy obstructive pulmonaryemphysema caseous pneumonia atelectasis miliary T B