acute hemorrhagic gastritis(2)pathogens: medicines (aspirin)/alcoholism2lesion:usually in fundus and body
(2) acute hemorrhagic gastritis 1）pathogens: medicines ( aspirin )/alcoholism 2）lesion: usually in fundus and body
(3)acutecorrosivegastritispathogen: engulfacid/baselesion: stomach wall necrosis →perforation
(3) acute corrosive gastritis 1）pathogen：engulf acid/ base 2）lesion：stomach wall necrosis →perforation
acuteinfective gastritis4pathogens: purulent bacteriumlesions: acute phlegmonous inflammation
(4) acute infective gastritis 1）pathogens: purulent bacterium 2）lesions: acute phlegmonous inflammation
2. chronic gastritischronic mucosal inflammatorychanges leading eventually to mucosal atrophy andepithelial metaplasia.PathogenesisHP chronic infectionchronic irritationH.pylori.A Steiner silver stain demonstrates the numerous darkly stainedautoimmuneHelicobacter organisms along the luminal surface of the gastric epitheliacells and within adherent mucus. Note that there is no tissue invasion bybacteriareflex ofbilious duodenal secretion
2. chronic gastritis—chronic mucosal inflammatory changes leading eventually to mucosal atrophy and epithelial metaplasia. Pathogenesis HP chronic infection chronic irritation autoimmune reflex of bilious duodenal secretion
INFECTIONCHRONICGASTRITISDamaging influences:AtrophyHelicobacterpyionIntestinalmetaplasiaLymphoid aggregatesureasetoxinsNeutrophil infiitratesGastric acidityPepticenzymesH.pyloriIntestinalmetaplasiaMucusNeutrophilsMucosaMuscularismucosaeSubmucosa