Parietal (oxyntic)cellsChief(zymogen)cellsEndocrinecellsMucin-secretingcellsFundusShorttubularCardiaglandsenCARDIACMUCOSABODY-FUNDICMUCOSABodyLefPylorusDOPyloricantrumvatGreatercuryD2D3D4MucouscellsLongtubularglandsPYLORIC MUCOSAFIGURE18.5Anatomical subdivisions of the stomach correlated with histological appearance of gastric mucosa in different regionsD1.D2,D3andD4arethefirsttofourthpartsoftheduodenum
8.1.2GASTRITISTABLE18.2:Classification of GastritisA.ACUTE GASTRITIS1.Acute H.pylori gastritis2.Other acute infective gastritis bacteria, viruses, fungi,parasites)3.Acute non-infective gastritisB. CHRONIC GASTRITIS:Body-fundic predominant1.TypeA (autoimmune)2.TypeB(H.pylori-related):Antral-predominantgastritis3.Type AB(environmental):Antral-bodygastritis4.Chemical (reflux)gastritis:Antral-bodypredominant5.Uncommon forms ofgastritis
8.1.2 GASTRITIS
inflammation of gastric mucosaacutegastritispathogens clearacute inflammation (neutrophilschronicgastritisautoimmunebilious duodenal secretion reflexHPinfection
inflammation of gastric mucosa acute gastritis pathogens clear acute inflammation (neutrophils ) chronic gastritis autoimmune bilious duodenal secretion reflex HP infection
l.acutegastritisDefinition: acute inflammation, erosion, and hemorrhageof the gastric mucosa due to a breakdown of themucosal barrier and acid-induced injuryEtiology: chronic aspirin or NSAID use/ Alcohol use/Smoking/ Postsurgery/ Burns/ Ischemia/ Stress/Uremia/ ChemotherapyPresentationEpigastric abdominal painGastric hemorrhage, hematemesis, and melena
1. acute gastritis Definition: acute inflammation, erosion, and hemorrhage of the gastric mucosa due to a breakdown of the mucosal barrier and acid-induced injury Etiology: chronic aspirin or NSAID use/ Alcohol use/ Smoking/ Postsurgery/ Burns/ Ischemia/ Stress/ Uremia/ Chemotherapy Presentation Epigastric abdominal pain Gastric hemorrhage, hematemesis, and melena
()acuteirritatedgastritispathogens: diet2) lesions: edema,hyperemia, hypersection of mucus, erosion
(1) acute irritated gastritis 1）pathogens：diet 2）lesions：edema, hyperemia, hypersection of mucus, erosion