RETINAL ARTERY OCCLUSION CRAO
RETINAL ARTERY OCCLUSION CRAO
CENTRAL RETINAL ARTERY OCCLUSION Real ocular emergency a Etiology Atherosclerosis Embolus Pale retina Embolus (plaque) Carotid or cardiac sources Systemic Diseases a Giant cell arteritis, disorders in coagulation, trauma, inflammatory or infectious diseases
CENTRAL RETINAL ARTERY OCCLUSION Real ocular emergency Etiology – Atherosclerosis – Embolus Carotid or cardiac sources – Systemic Diseases Giant cell arteritis, disorders in coagulation, trauma, inflammatory or infectious diseases
CENTRAL RETINAL ARTERY OCCLUSION Clinical manifestation Unilateral painless catastrophic visual loss NLP-CF Disappeared direct response to light in affected eye Opacified retinal edema, cherry-red spot in foveolar, thin retinal artery Several weeks later: resolved retinal edema, pale optic disc, vessels reducing to thin white lines
CENTRAL RETINAL ARTERY OCCLUSION Clinical Manifestation – Unilateral painless catastrophic visual loss NLP-CF – Disappeared direct response to light in affected eye – Opacified retinal edema, cherry-red spot in foveolar, thin retinal artery – Several weeks later: resolved retinal edema, pale optic disc, vessels reducing to thin white lines
CENTRAL RETINAL ARTERY OCCLUSION t treatment Controlling intraocular pressure Eye ball massage, Diamox, anterior chamber paracentesis Retinal vasodilation Taking Nitroglycerin or amylnitrate under the tongue a Benzoxazoline 12.5-25mg retrobulbar Injection ■ Intravenous use Inhaling 95%oO2 and 5%o CO2 mixture Examining other systemic risk factors
CENTRAL RETINAL ARTERY OCCLUSION Treatment – Controlling intraocular pressure Eye ball massage, Diamox, anterior chamber paracentesis – Retinal vasodilation Taking Nitroglycerin or amylnitrate under the tongue Benzoxazoline 12.5-25mg retrobulbar Injection Intravenous use – Inhaling 95% O2 and 5% CO2 mixture – Examining other systemic risk factors