A CA M. C.A. PGA INTERNAL CAROTID A AY-VERTEBRAL A
Anatomy and pathology Occlusion leads to sudden severe ischaemia in the area of brain tissue supplied by the occluded artery, and recovery depends upon rapid lysis or fragmentation of the occluding material Reversal of neurological function within minutes or hours gives rise to the clinical picture of a transient ischaemic attack
Anatomy and pathology Occlusion leads to sudden severe ischaemia in the area of brain tissue supplied by the occluded artery, and recovery depends upon rapid lysis or fragmentation of the occluding material: Reversal of neurological function within minutes or hours gives rise to the clinical picture of a transient ischaemic attack
Anatomy and pathology When the neurological deficit lasts longer than 24 hours, it may be called a reversible ischaemic neurological deficit RIND ) if it recovers completely in a few days or a completed stroke if there is a persistent deficit Sometimes recovery is very slow and incomplete
Anatomy and pathology When the neurological deficit lasts longer than 24 hours, it may be called a reversible ischaemic neurological deficit ( RIND ) if it recovers completely in a few days, or a completed stroke if there is a persistent deficit. Sometimes recovery is very slow and incomplete
Neurological symptoms and signs The loss of function that the patient notices, and which may be apparent on examination, entirely depends on the area of brain tissue involved in the ischaemic process
Neurological symptoms and signs The loss of function that the patient notices, and which may be apparent on examination, entirely depends on the area of brain tissue involved in the ischaemic process
A CA M. C.A. PGA INTERNAL CAROTID A AY-VERTEBRAL A