Clinical Acute carbon monoxide poisoning Mild poisoning · COHb level:10%-20% Headache, nausea, vomiting, fatigue Moderate poisoning · COHb leve|:30%-40% Confusion, Agitation hallucination Visual disturbance syncope, seIzure Skin: Classic cherry red skin is rare (ie, When you're cherry red, you're dead); pallor is present more often Severe poisoning ·COHb|eve|:40%-60% Noncardiogenic pulmonary edema, arrhythmia Papilledema
Clinical ◼ Acute carbon monoxide poisoning – Mild poisoning • COHb level :10%-20%. • Headache, nausea, vomiting, fatigue. – Moderate poisoning • COHb level :30%-40%. • Confusion, Agitation, Hallucination, Visual disturbance, syncope, seizure • Skin: Classic cherry red skin is rare (ie, “When you're cherry red, you're dead”); pallor is present more often. – Severe poisoning • COHb level :40%-60%. • Noncardiogenic pulmonary edema, arrhythmia, Papilledema
Clinical a delayed neuropsychiatric symptoms Long-term exposures or severe acute exposures frequently result in long-term neuropsychiatric sequelae. Additionally, some individuals develop delayed neuropsychiatric symptoms, often after severe intoxications associated with coma(about 3%-10%of all patients After 2-60days" normal period, chronic headaches, memory problems, and parkinsonian type tremor, re-occur
◼ delayed neuropsychiatric symptoms – Long-term exposures or severe acute exposures frequently result in long-term neuropsychiatric sequelae. Additionally, some individuals develop delayed neuropsychiatric symptoms, often after severe intoxications associated with coma (about 3%-10% of all patients) – After 2-60days “normal period” , chronic headaches, memory problems, and parkinsoniantype tremor, re-occur . Clinical